Neuroplasticity, also known as brain plasticity, neuroelasticity, or neural plasticity, is the ability of the brain to change continuously throughout an individual's life.
This is possible because brain activity associated with a given function can be transferred to a different location, the proportion of grey matter can change, or it can weaken and strengthen or weaken over time.
The brain has an innate ability to heal itself, and to compensate for lost function by strengthening existing pathways, or recruiting new pathways should an existing pathway be irrevocably damaged.
There are a large number of mechanisms contributing to this ability, which are collectively termed neuroplasticity, and are described below. 
Human brain inside a head made in 3d over a blue background


Structural Plasticity
Neurons themselves can change structurally. Requires neighbouring neurons to fire in synchrony. Receiving (postsynaptic) neuron can increase the length and number of dendrites (the branches of the neuron which pick up incoming signals). Transmitting neuron can create more axonal branches and terminals.


Synaptic Plasticity
Neuroplasticity can occur at the terminal between two neurons. When a neuron is repeatedly active, the amount of neurotransmitter released for each impulse (action potential) increases. The number of receptors on the receiving neuron can also increase.


Communication between neurons is critically dependent on the insulation of the wiring (axon) Axon is covered in myelin. Speed of electric signal dependent on amount of myelin. Use of a pathway increases the amount of myelin.


Thousands of new neurons are produced each day from stem cells. In the event of injury, stem cells migrate from their origin in the brain to damaged areas. It is not yet known whether and under what conditions these stem cells can repair damaged tissues.

We cannot say which of the above mechanisms are specifically targeted by the REVIVER device, but any alteration to brain function will be a result of one or multiple of the above mechanisms.


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